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April 2002

By James R. Roberts, MD

 

A 60-year-old man sought medical attention in the emergency department for painless elbow swelling that had developed gradually over the previous week. He could not remember falling or any other precipitating trauma. Examination revealed a painless, fluctuant, mobile, homogeneous mass about the size of a golf ball over the olecranon. The skin was intact; no warmth or tenderness was noted, and the joint had full range of motion. The patient was afebrile. Radiographs revealed no evidence of fracture, foreign body, or joint effusion. Though his medical history documented only well-controlled hypertension and no current anticoagulant use, the patient thought he remembered having the same problem 15 years ago and "getting a shot for it." Most of his afternoons, he did admit, were spent with his elbows on a bar as he chatted with friends over a "few beers." That revelation led one examiner to assume that the elbow mass was probably a bruise or other trauma sustained while the patient was intoxicated and that it should be treated with an elastic bandage and some ibuprofen. A logical conclusion, it would seem, but the examiner was overlooking another possibility.

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The actual diagnosis was an accumulation of sterile fluid in the olecranon bursa, of which this was a classic example. Though neither painful nor acutely inflammatory, the disorder is nevertheless referred to as olecranon bursitis. The exact etiology remains unknown in many cases, but generally the condition has been ascribed to minor or repeated trauma to the elbow. Radiographs are usually obtained because patients often conjure up some type of trauma in the history to explain the swelling. A calcific spur on the olecranon is a common finding but has no diagnostic or etiologic significance.
     A more serious variant of olecranon bursal fluid accumulation is septic olecranon bursitis, which is usually caused by gram-positive bacteria such as Staphylococcus aureus. This is easily distinguished from benign sterile fluid accumulation by significant pain and tenderness, more diffuse swelling, and redness and warmth of the skin over the area. It is often accompanied by fever and leukocytosis. (Acute gouty bursitis, however, may mimic a bursal infection.) Septic bursitis usually occurs in patients with a predisposing factor such as diabetes or a break in the skin. Drainage and antibiotics, occasionally by parenteral administration, are required. A final consideration in the differential diagnosis of this case was hematoma, which would have been likely if the patient had been taking coumadin.
     The patient would likely have returned soon if misdiagnosed, since spontaneous resolution of benign olecranon bursitis is unusual. The fluid needs to be aspirated with an 18-gauge needle (left), followed by injection of a long-acting corticosteroid, such as 40 mg methylprednisolone, via the same needle (right). Without the steroid injection, the fluid typically reaccumulates. After the procedure, an elastic compression bandage can be applied; usually there is no need for analgesic or anti-inflammatory medication. Culture and additional analysis of the aspirated fluid rarely provide any useful information and is not standard if infection can be ruled out on clinical grounds.

Emerg Med 34(4):55, 2002

Dr. Roberts is professor of emergency medicine at the Medical College of Pennsylvania and chairman of the department of emergency medicine at Mercy Catholic Medical Center in Philadelphia. He is also a member of the Emergency Medicine editorial board.



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